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Showing 1 to 12 of 629 entries
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Erratum to: Intrauterine Growth Retardation (IUGR) as a Novel Condition of Insulin-Like Growth Factor-1 (IGF-1) Deficiency.

Reviews of physiology, biochemistry and pharmacology

Martín-Estal I, de la Garza RG, Castilla-Cortázar I.
PMID: 27012750
Rev Physiol Biochem Pharmacol. 2016;170:129. doi: 10.1007/112_2016_1.

No abstract available.

Cite
Martín-Estal I, de la Garza RG, Castilla-Cortázar I. Erratum to: Intrauterine Growth Retardation (IUGR) as a Novel Condition of Insulin-Like Growth Factor-1 (IGF-1) Deficiency. Rev Physiol Biochem Pharmacol. 2016;170:129doi: 10.1007/112_2016_1.
Martín-Estal, I., de la Garza, R. G., & Castilla-Cortázar, I. (2016). Erratum to: Intrauterine Growth Retardation (IUGR) as a Novel Condition of Insulin-Like Growth Factor-1 (IGF-1) Deficiency. Reviews of physiology, biochemistry and pharmacology, 170129. https://doi.org/10.1007/112_2016_1
Martín-Estal, I, et al. "Erratum to: Intrauterine Growth Retardation (IUGR) as a Novel Condition of Insulin-Like Growth Factor-1 (IGF-1) Deficiency." Reviews of physiology, biochemistry and pharmacology vol. 170 (2016): 129. doi: https://doi.org/10.1007/112_2016_1
Martín-Estal I, de la Garza RG, Castilla-Cortázar I. Erratum to: Intrauterine Growth Retardation (IUGR) as a Novel Condition of Insulin-Like Growth Factor-1 (IGF-1) Deficiency. Rev Physiol Biochem Pharmacol. 2016;170:129. doi: 10.1007/112_2016_1. PMID: 27012750.
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Retraction notice to “Cyclodextrin-complexed curcumin exhibits anti-inflammatory and antiproliferative activities superior to those of curcumin through higher cellular uptake” [Biochem. Pharmacol. 80 (2010) 1021–1032].

Biochemical pharmacology

[No authors listed]
PMID: 26985464
Biochem Pharmacol. 2016 Feb 15;102:142. doi: 10.1016/j.bcp.2015.11.007. Epub 2016 Feb 20.

No abstract available.

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Retraction notice to “Cyclodextrin-complexed curcumin exhibits anti-inflammatory and antiproliferative activities superior to those of curcumin through higher cellular uptake” [Biochem. Pharmacol. 80 (2010) 1021–1032]. Biochem Pharmacol. 2016;102:142doi: 10.1016/j.bcp.2015.11.007.
(2016). Retraction notice to “Cyclodextrin-complexed curcumin exhibits anti-inflammatory and antiproliferative activities superior to those of curcumin through higher cellular uptake” [Biochem. Pharmacol. 80 (2010) 1021–1032]. Biochemical pharmacology, 102142. https://doi.org/10.1016/j.bcp.2015.11.007
"Retraction notice to “Cyclodextrin-complexed curcumin exhibits anti-inflammatory and antiproliferative activities superior to those of curcumin through higher cellular uptake” [Biochem. Pharmacol. 80 (2010) 1021–1032]." Biochemical pharmacology vol. 102 (2016): 142. doi: https://doi.org/10.1016/j.bcp.2015.11.007
Retraction notice to “Cyclodextrin-complexed curcumin exhibits anti-inflammatory and antiproliferative activities superior to those of curcumin through higher cellular uptake” [Biochem. Pharmacol. 80 (2010) 1021–1032]. Biochem Pharmacol. 2016 Feb 15;102:142. doi: 10.1016/j.bcp.2015.11.007. Epub 2016 Feb 20. PMID: 26985464; PMCID: PMC4801114.
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BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN.

Biochemical pharmacology

Breese GR, Cooper BR, Smith RD.
PMID: 21394240
Biochem Pharmacol. 1974 Apr;23:574-579. doi: 10.1016/0006-2952(74)90086-0.

No abstract available.

Cite
Breese GR, Cooper BR, Smith RD. BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN. Biochem Pharmacol. 1974;23:574-579doi: 10.1016/0006-2952(74)90086-0.
Breese, G. R., Cooper, B. R., & Smith, R. D. (1974). BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN. Biochemical pharmacology, 23574-579. https://doi.org/10.1016/0006-2952(74)90086-0
Breese, George R, et al. "BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN." Biochemical pharmacology vol. 23 (1974): 574-579. doi: https://doi.org/10.1016/0006-2952(74)90086-0
Breese GR, Cooper BR, Smith RD. BIOCHEMICAL AND BEHAVIOURAL ALTERATIONS FOLLOWING 6-HYDROXYDOPAMINE ADMINISTRATION INTO BRAIN. Biochem Pharmacol. 1974 Apr;23:574-579. doi: 10.1016/0006-2952(74)90086-0. PMID: 21394240; PMCID: PMC3050642.
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Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice.

Journal of biochemical and pharmacological research

Bai X, Fermandez E, Gould G, Strong R.
PMID: 24494173
J Biochem Pharmacol Res. 2013 Dec 01;1(4):228-235.

Much evidence suggests that oxidative stress plays a role in schizophrenia pathogenesis. Major oxidative stress sources include hydrogen peroxide and biogenic aldehydes that are mainly cleared

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Bai X, Fermandez E, Gould G, et al. Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. J Biochem Pharmacol Res. 2013;1(4):228-235
Bai, X., Fermandez, E., Gould, G., & Strong, R. (2013). Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. Journal of biochemical and pharmacological research, 1(4), 228-235.
Bai, Xiang, et al. "Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice." Journal of biochemical and pharmacological research vol. 1,4 (2013): 228-235.
Bai X, Fermandez E, Gould G, Strong R. Homozygous Deletion of Glutathione Peroxidase 1 and Aldehyde Dehydrogenase 1a1 Genes Is Not Associated with Schizophrenia-Like Behavior in Mice. J Biochem Pharmacol Res. 2013 Dec 01;1(4):228-235. PMID: 24494173; PMCID: PMC3909525.
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Natural Polyphenols Inhibit Lysine-Specific Demethylase-1 .

Journal of biochemical and pharmacological research

Abdulla A, Zhao X, Yang F.
PMID: 23662249
J Biochem Pharmacol Res. 2013 Mar 01;1(1):56-63.

Natural polyphenols, such as resveratrol, have beneficial functions on major human diseases such as cancer, diabetes, and cardiovascular disease. Besides acting as antioxidants, some of these polyphenols can also target proteins to modulate specific biological pathways. The lysine-specific histone...

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Abdulla A, Zhao X, Yang F. Natural Polyphenols Inhibit Lysine-Specific Demethylase-1 . J Biochem Pharmacol Res. 2013;1(1):56-63
Abdulla, A., Zhao, X., & Yang, F. (2013). Natural Polyphenols Inhibit Lysine-Specific Demethylase-1 . Journal of biochemical and pharmacological research, 1(1), 56-63.
Abdulla, Arian, et al. "Natural Polyphenols Inhibit Lysine-Specific Demethylase-1 ." Journal of biochemical and pharmacological research vol. 1,1 (2013): 56-63.
Abdulla A, Zhao X, Yang F. Natural Polyphenols Inhibit Lysine-Specific Demethylase-1 . J Biochem Pharmacol Res. 2013 Mar 01;1(1):56-63. PMID: 23662249; PMCID: PMC3646376.
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Corrigendum to "Let-7b ameliorates Crohn's disease-associated adherent-invasive E coli induced intestinal inflammation via modulating Toll-Like Receptor 4 expression in intestinal epithelial cells" [Biochem. Pharmacol. 15 (2018) 196-203].

Biochemical pharmacology

Guo Z, Cai X, Guo X, Xu Y, Gong J, Li Y, Zhu W.
PMID: 30292943
Biochem Pharmacol. 2018 Dec;158:35-36. doi: 10.1016/j.bcp.2018.09.025. Epub 2018 Oct 04.

No abstract available.

Cite
Guo Z, Cai X, Guo X, et al. Corrigendum to "Let-7b ameliorates Crohn's disease-associated adherent-invasive E coli induced intestinal inflammation via modulating Toll-Like Receptor 4 expression in intestinal epithelial cells" [Biochem. Pharmacol. 15 (2018) 196-203]. Biochem Pharmacol. 2018;158:35-36doi: 10.1016/j.bcp.2018.09.025.
Guo, Z., Cai, X., Guo, X., Xu, Y., Gong, J., Li, Y., & Zhu, W. (2018). Corrigendum to "Let-7b ameliorates Crohn's disease-associated adherent-invasive E coli induced intestinal inflammation via modulating Toll-Like Receptor 4 expression in intestinal epithelial cells" [Biochem. Pharmacol. 15 (2018) 196-203]. Biochemical pharmacology, 15835-36. https://doi.org/10.1016/j.bcp.2018.09.025
Guo, Zhen, et al. "Corrigendum to "Let-7b ameliorates Crohn's disease-associated adherent-invasive E coli induced intestinal inflammation via modulating Toll-Like Receptor 4 expression in intestinal epithelial cells" [Biochem. Pharmacol. 15 (2018) 196-203]." Biochemical pharmacology vol. 158 (2018): 35-36. doi: https://doi.org/10.1016/j.bcp.2018.09.025
Guo Z, Cai X, Guo X, Xu Y, Gong J, Li Y, Zhu W. Corrigendum to "Let-7b ameliorates Crohn's disease-associated adherent-invasive E coli induced intestinal inflammation via modulating Toll-Like Receptor 4 expression in intestinal epithelial cells" [Biochem. Pharmacol. 15 (2018) 196-203]. Biochem Pharmacol. 2018 Dec;158:35-36. doi: 10.1016/j.bcp.2018.09.025. Epub 2018 Oct 04. PMID: 30292943.
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Erratum to "Inhibition of botulinum neurotoxins interchain disulfide bond reduction prevents the peripheral neuroparalysis of botulism" [Biochem. Pharmacol. 98 (2015) 522-530].

Biochemical pharmacology

Zanetti G, Azarnia Tehran D, Pirazzini M, Binz T, Shone CC, Fillo S, Lista F, Rossetto O, Montecucco C.
PMID: 27498956
Biochem Pharmacol. 2016 Sep 01;115:152. doi: 10.1016/j.bcp.2016.07.006. Epub 2016 Jul 12.

No abstract available.

Cite
Zanetti G, Azarnia Tehran D, Pirazzini M, et al. Erratum to "Inhibition of botulinum neurotoxins interchain disulfide bond reduction prevents the peripheral neuroparalysis of botulism" [Biochem. Pharmacol. 98 (2015) 522-530]. Biochem Pharmacol. 2016;115:152doi: 10.1016/j.bcp.2016.07.006.
Zanetti, G., Azarnia Tehran, D., Pirazzini, M., Binz, T., Shone, C. C., Fillo, S., Lista, F., Rossetto, O., & Montecucco, C. (2016). Erratum to "Inhibition of botulinum neurotoxins interchain disulfide bond reduction prevents the peripheral neuroparalysis of botulism" [Biochem. Pharmacol. 98 (2015) 522-530]. Biochemical pharmacology, 115152. https://doi.org/10.1016/j.bcp.2016.07.006
Zanetti, Giulia, et al. "Erratum to "Inhibition of botulinum neurotoxins interchain disulfide bond reduction prevents the peripheral neuroparalysis of botulism" [Biochem. Pharmacol. 98 (2015) 522-530]." Biochemical pharmacology vol. 115 (2016): 152. doi: https://doi.org/10.1016/j.bcp.2016.07.006
Zanetti G, Azarnia Tehran D, Pirazzini M, Binz T, Shone CC, Fillo S, Lista F, Rossetto O, Montecucco C. Erratum to "Inhibition of botulinum neurotoxins interchain disulfide bond reduction prevents the peripheral neuroparalysis of botulism" [Biochem. Pharmacol. 98 (2015) 522-530]. Biochem Pharmacol. 2016 Sep 01;115:152. doi: 10.1016/j.bcp.2016.07.006. Epub 2016 Jul 12. PMID: 27498956.
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A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease.

Journal of biochemical and pharmacological research

Bai X, Hussong SA.
PMID: 26405617
J Biochem Pharmacol Res. 2014 Jun;2(2):54-56.

No abstract available.

Cite
Bai X, Hussong SA. A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease. J Biochem Pharmacol Res. 2014;2(2):54-56
Bai, X., & Hussong, S. A. (2014). A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease. Journal of biochemical and pharmacological research, 2(2), 54-56.
Bai, Xiang, and Hussong, Stacy A. "A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease." Journal of biochemical and pharmacological research vol. 2,2 (2014): 54-56.
Bai X, Hussong SA. A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease. J Biochem Pharmacol Res. 2014 Jun;2(2):54-56. PMID: 26405617; PMCID: PMC4578241.
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Progress in Making Ras as a Druggable Target.

Biochemistry & pharmacology : open access

Bhattacharyya S.
PMID: 26491623
Biochem Pharmacol (Los Angel). 2014 Apr 01;3(2). doi: 10.4172/2167-0501.1000e157. Epub 2014 Mar 14.

No abstract available.

Cite
Bhattacharyya S. Progress in Making Ras as a Druggable Target. Biochem Pharmacol (Los Angel). 2014;3(2)doi: 10.4172/2167-0501.1000e157.
Bhattacharyya, S. (2014). Progress in Making Ras as a Druggable Target. Biochemistry & pharmacology : open access, 3(2), . https://doi.org/10.4172/2167-0501.1000e157
Bhattacharyya, Sibaprasad. "Progress in Making Ras as a Druggable Target." Biochemistry & pharmacology : open access vol. 3,2 (2014). doi: https://doi.org/10.4172/2167-0501.1000e157
Bhattacharyya S. Progress in Making Ras as a Druggable Target. Biochem Pharmacol (Los Angel). 2014 Apr 01;3(2). doi: 10.4172/2167-0501.1000e157. Epub 2014 Mar 14. PMID: 26491623; PMCID: PMC4610733.
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Corrigendum to 'Effects of 17β-estradiol on colorectal cancer development after azoxymethane/dextran sulfate sodium treatment of ovariectomized mice' [Biochem. Pharmacol. 164 (2019) 139-151].

Biochemical pharmacology

Song CH, Kim N, Min Lee S, Hee Nam R, In Choi S, Ra Kang S, Shin E, Ho Lee D, Lee HN, Surh YJ.
PMID: 31377420
Biochem Pharmacol. 2019 Oct;168:339-340. doi: 10.1016/j.bcp.2019.07.020. Epub 2019 Aug 01.

No abstract available.

Cite
Song CH, Kim N, Min Lee S, et al. Corrigendum to 'Effects of 17β-estradiol on colorectal cancer development after azoxymethane/dextran sulfate sodium treatment of ovariectomized mice' [Biochem. Pharmacol. 164 (2019) 139-151]. Biochem Pharmacol. 2019;168:339-340doi: 10.1016/j.bcp.2019.07.020.
Song, C. H., Kim, N., Min Lee, S., Hee Nam, R., In Choi, S., Ra Kang, S., Shin, E., Ho Lee, D., Lee, H. N., & Surh, Y. J. (2019). Corrigendum to 'Effects of 17β-estradiol on colorectal cancer development after azoxymethane/dextran sulfate sodium treatment of ovariectomized mice' [Biochem. Pharmacol. 164 (2019) 139-151]. Biochemical pharmacology, 168339-340. https://doi.org/10.1016/j.bcp.2019.07.020
Song, Chin-Hee, et al. "Corrigendum to 'Effects of 17β-estradiol on colorectal cancer development after azoxymethane/dextran sulfate sodium treatment of ovariectomized mice' [Biochem. Pharmacol. 164 (2019) 139-151]." Biochemical pharmacology vol. 168 (2019): 339-340. doi: https://doi.org/10.1016/j.bcp.2019.07.020
Song CH, Kim N, Min Lee S, Hee Nam R, In Choi S, Ra Kang S, Shin E, Ho Lee D, Lee HN, Surh YJ. Corrigendum to 'Effects of 17β-estradiol on colorectal cancer development after azoxymethane/dextran sulfate sodium treatment of ovariectomized mice' [Biochem. Pharmacol. 164 (2019) 139-151]. Biochem Pharmacol. 2019 Oct;168:339-340. doi: 10.1016/j.bcp.2019.07.020. Epub 2019 Aug 01. PMID: 31377420.
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Calcium-Permeable Channels in Tumor Vascularization: Peculiar Sensors of Microenvironmental Chemical and Physical Cues.

Reviews of physiology, biochemistry and pharmacology

Scarpellino G, Munaron L, Cantelmo AR, Fiorio Pla A.
PMID: 32809072
Rev Physiol Biochem Pharmacol. 2020 Aug 19; doi: 10.1007/112_2020_32. Epub 2020 Aug 19.

Calcium (Ca

Cite
Scarpellino G, Munaron L, Cantelmo AR, et al. Calcium-Permeable Channels in Tumor Vascularization: Peculiar Sensors of Microenvironmental Chemical and Physical Cues. Rev Physiol Biochem Pharmacol. 2020;doi: 10.1007/112_2020_32.
Scarpellino, G., Munaron, L., Cantelmo, A. R., & Fiorio Pla, A. (2020). Calcium-Permeable Channels in Tumor Vascularization: Peculiar Sensors of Microenvironmental Chemical and Physical Cues. Reviews of physiology, biochemistry and pharmacology, . https://doi.org/10.1007/112_2020_32
Scarpellino, Giorgia, et al. "Calcium-Permeable Channels in Tumor Vascularization: Peculiar Sensors of Microenvironmental Chemical and Physical Cues." Reviews of physiology, biochemistry and pharmacology vol. (2020). doi: https://doi.org/10.1007/112_2020_32
Scarpellino G, Munaron L, Cantelmo AR, Fiorio Pla A. Calcium-Permeable Channels in Tumor Vascularization: Peculiar Sensors of Microenvironmental Chemical and Physical Cues. Rev Physiol Biochem Pharmacol. 2020 Aug 19; doi: 10.1007/112_2020_32. Epub 2020 Aug 19. PMID: 32809072.
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Corrigendum to "Concurrent MEK and autophagy inhibition is required to restore cell death associated danger-signalling in Vemurafenib-resistant melanoma cells" [Biochem. Pharmacol. 93 (3) (2015) 290-304].

Biochemical pharmacology

Martin S, Dudek-Perić AM, Maes H, Garg AD, Gabrysiak M, Demirsoy S, Swinnen JV, Agostinis P.
PMID: 32085947
Biochem Pharmacol. 2020 May;175:113810. doi: 10.1016/j.bcp.2020.113810. Epub 2020 Feb 19.

No abstract available.

Cite
Martin S, Dudek-Perić AM, Maes H, et al. Corrigendum to "Concurrent MEK and autophagy inhibition is required to restore cell death associated danger-signalling in Vemurafenib-resistant melanoma cells" [Biochem. Pharmacol. 93 (3) (2015) 290-304]. Biochem Pharmacol. 2020;175:113810doi: 10.1016/j.bcp.2020.113810.
Martin, S., Dudek-Perić, A. M., Maes, H., Garg, A. D., Gabrysiak, M., Demirsoy, S., Swinnen, J. V., & Agostinis, P. (2020). Corrigendum to "Concurrent MEK and autophagy inhibition is required to restore cell death associated danger-signalling in Vemurafenib-resistant melanoma cells" [Biochem. Pharmacol. 93 (3) (2015) 290-304]. Biochemical pharmacology, 175113810. https://doi.org/10.1016/j.bcp.2020.113810
Martin, S, et al. "Corrigendum to "Concurrent MEK and autophagy inhibition is required to restore cell death associated danger-signalling in Vemurafenib-resistant melanoma cells" [Biochem. Pharmacol. 93 (3) (2015) 290-304]." Biochemical pharmacology vol. 175 (2020): 113810. doi: https://doi.org/10.1016/j.bcp.2020.113810
Martin S, Dudek-Perić AM, Maes H, Garg AD, Gabrysiak M, Demirsoy S, Swinnen JV, Agostinis P. Corrigendum to "Concurrent MEK and autophagy inhibition is required to restore cell death associated danger-signalling in Vemurafenib-resistant melanoma cells" [Biochem. Pharmacol. 93 (3) (2015) 290-304]. Biochem Pharmacol. 2020 May;175:113810. doi: 10.1016/j.bcp.2020.113810. Epub 2020 Feb 19. PMID: 32085947.
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Showing 1 to 12 of 629 entries